Rejected
at 2:02 a.m. Nov, 01, 2023 ]
by
dollajas
Author:
learnwell
Type of change:
Updated content
Rationale for change
Added relevant cloze hints for quick review via pertinent decision making and classification without giving away the answer.
Rejection reason
agree. high yield
Current
Suggested
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Extra
- Normally in lung injury, type I pneumocytes are destroyed, type II pneumocytes undergo hyperplasia to make cells that differentiate into and replace the type I, then remaining type II die
- In IPF, chronic injury of type II pneumocytes leads to constant hyperplasia of type II pneumocytes (increased), which produce TGF-β that stimulates activity / proliferation of fibroblasts (increased); while type I pneumocytes die off (decrease) / are replaced by fibrotic tissue
- In IPF, chronic injury of type II pneumocytes leads to constant hyperplasia of type II pneumocytes (increased), which produce TGF-β that stimulates activity / proliferation of fibroblasts (increased); while type I pneumocytes die off (decrease) / are replaced by fibrotic tissue
Lecture Notes
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Missed Questions
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Pathoma
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Boards and Beyond
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First Aid
Sketchy 2
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Sketchy Extra
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Picmonic
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Pixorize
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Physeo
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OME
Additional Resources
IPF seen on PATHOLOGY may also be referred to as usual interstitial pneumonia (UIP) 
- normally in lung injury, type I pneumocytes are destroyed, type II pneumocytes undergo hyperplasia to make cells that differentiate into and replace the type I, then remaining type II die
- in IPF, chronic injury of type II pneumocytes leads to constant hyperplasia of type II pneumocytes (increased), which produce TGF-β that stimulates activity / proliferation of fibroblasts (increased); while type I pneumocytes die off (decrease) / are replaced by fibrotic tissue
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