Accepted
at 2:38 p.m. Nov, 29, 2024
by
andrewmathias8
Author:
Brian_BH
Type of change:
Other
Rationale for change
Removed formatting from cloze brackets which can cause display issues.
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Due to macrophage-mediated structural degradation; results in increased O2 sat and pressure in the RV
| Timeline | Complication | Extra |
|---|---|---|
| Days to months | Arrhythmia | Common cause of death post-MI |
| 1-3 days | Fibrinous pericarditis | Friction rub |
| < 1 week | Papillary muscle rupture | Posteromedial, mitral regurgitation |
| Interventricular wall rupture | VSD, ↑O2 sat in the RV | |
| < 2 weeks | Ventricular pseudoaneurysm | Contained within the pericardium |
| Ventricular free wall rupture | Cardiac tamponade | |
| 2 weeks - months | True ventricular aneurysm | Contains all 3 layers |
| Dressler syndrome | Autoimmune mediated fibrinous pericarditis |
Lecture Notes
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Missed Questions
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Pathoma
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Boards and Beyond
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First Aid
Sketchy 2
Sketchy Extra
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Picmonic
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Pixorize
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Physeo
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Bootcamp
OME
Additional Resources
Subaortic ventricular septal defect
Gross pathology of the heart (longitudinal section through the left ventricle)
An oval hole with a diameter of ∼1 cm (2) can be seen on the upper left margin of the ventricular septum (1) just below the aorta (3). The aorta has been incised and is recognizable by its pale, smooth structure and the way it branches off into the coronary arteries (circles). The mitral valves (4) with the papillary muscles and chordae tendineae (5) can be seen on the right.
The location of the hole within the septum is characteristic of subaortic ventricular septal defect.
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