Created at 3:05 p.m. May, 01, 2024
Author: GroupG_Strep93
Related Note: 1481771819827 1
Rationale for change

resuggestions as this has been rejected long time ago.

lipohyalinosis is specifically associated with brain aneurysms as lipids play a role in the pathogenesis. saying hyaline arteriolosclerosis would essentially exclude the lipofibrous deposition in the vessel wall because lipids do not really play a role in hyaline arteriolosclerosis. this is backed up by Extra, uworld and AMBOSS. Although FirstAid 24 p. 526 also mentions hyaline arteriosclerosis (not arteriolosclerosis) it specifies lipohyalinosis
Source: AMBOSS - https://next.amboss.com/us/article/iJ0JFS#L2ebfa6cf0d6cd25276c580d257789565

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Extra
The primary causes of lacunar infarcts are:

- Lipohyalinosis occurs secondary to leakage of plasma proteins through damaged endothelium and is characterized by hyaline thickening of the vascular wall, collagenous sclerosis, and accumulation of mural foamy macrophages

- Microatheromas result from atherosclerotic accumulation of foamy macrophages within the intimal layer of a penetrating artery near its origin off the parent vessel

- These changes predispose to small vessel occlusion and infarction of CNS tissue with liquefactive necrosis and the formation of a fluid-filled cavity
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Watch associated Bootcamp video - Ischemic Cerebrovascular Accidents: Lacunar Infarction
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